BiochemProbe — Life Science Research Reagents

TRAM 39 free base · Synonyms: TRAM-39 · TRAM39

TRAM-39 is a selective blocker of intermediate conductance Ca2+-activated K+ channels.

For research use only. Not for human or veterinary use.

Purity >98% Stock Inquire

TRAM 39 structure

CAS No.: 197525-99-8

Download structure (.mol)

Pack sizes & pricing

Size Price SKU Stock
50 mg USD 420.00 BP-02436-50mg In stock Get quote
100 mg USD 670.00 BP-02436-100mg In stock Get quote
250 mg USD 1,340.00 BP-02436-250mg In stock Get quote
500 mg USD 2,150.00 BP-02436-500mg In stock Get quote
1 g USD 3,440.00 BP-02436-1g In stock Get quote
2 g Inquire BP-02436-2g In stock Inquire
3 g Inquire BP-02436-3g In stock Inquire

Need another size or custom synthesis? Request a quote.

Description

TRAM-39 is a selective blocker of intermediate conductance Ca2+-activated K+ (IKCa) channels. TRAM-39 inhibits KCa3.1 channel with an IC50 value of 60 nM. TRAM-39 can be used for the research of ataxia, epilepsy, memory disorders, schizophrenia and Parkinson’s disease .

Biological activity

In Vitro TRAM-39 (1 μM) reduces the peak amplitude of gKCa2 in sympathetic LAH neurons (mean percentage change 56%) in BAE cells. TRAM-39 (200 nM) diminishes lipopolysaccharide (LPS)-induced cryptdin release from paneth cells. In Vivo TRAM-39 (1 μM; adds to the perfusate) fully blocks all IKCa channels in multicellular preparations.

Identifiers

SMILES
N#CC(C1=CC=CC=C1)(C2=CC=CC=C2)C3=CC=CC=C3Cl
InChIKey
JHNRTPKGSCVKKC-UHFFFAOYSA-N

Specifications

MW (average)
303.7850
Formula
C20H14ClN
CAS No.
197525-99-8
Physical state
Solid
Color
Off-white to light yellow
Shipping
Shipped under ambient temperature as non-hazardous chemical. This product is stable enough for a few weeks during ordinary shipping and time spent in Customs.
Storage
4°C, sealed storage, away from moisture

Solubility

Soluble in DMSO : 50 mg/mL

References

[1].Davies PJ, et al. Different types of potassium channels underlie the long afterhyperpolarization in guinea-pig sympathetic and enteric neurons. Auton Neurosci. 2006 Jan 30;124(1-2):26-30.
[2].Burnham MP, et al. Impaired small-conductance Ca2+-activated K+ channel-dependent EDHF responses in Type II diabetic ZDF rats. Br J Pharmacol. 2006 Jun;148(4):434-41.
[3].Ayabe T, et al. Modulation of mouse Paneth cell alpha-defensin secretion by mIKCa1, a Ca2+-activated, intermediate conductance potassium channel. J Biol Chem. 2002 Feb 1;277(5):3793-800.
[4].Wulff H, et al. Modulators of small- and intermediate-conductance calcium-activated potassium channels and their therapeutic indications. Curr Med Chem. 2007;14(13):1437-57.

Same research area

Search Neurological Disease